MIT researchers suggests a potential molecular strategy for treating fragile X syndrome by enhancing NMDA subunit function.
A new study shows that enhancing activity of a specific component of 'NMDA' receptors normalizes protein synthesis, neural activity and seizure susceptibility in the hippocampus of fragile X lab mice.
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AZoLifeSciences on MSNBreakthrough mRNA Technology Paves the Way for Precision MedicineImagine a breakthrough in cancer treatment where only malignant cells are targeted, sparing healthy host cells; or patients ...
Building on more than two decades of research, a study by MIT neuroscientists at The Picower Institute for Learning and Memory reports a new way to ...
MIT neuroscientists have made a breakthrough in treating fragile X syndrome by leveraging a novel neurotransmitter signaling ...
Imagine a breakthrough in cancer treatment where only malignant cells are targeted, sparing healthy host cells; or patients ...
Imagine a breakthrough in cancer treatment where only malignant cells are targeted, sparing healthy host cells; or patients with abnormal protein synthesis are treated to produce a healthy protein.
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Japanese researchers enhance mRNA cancer drugs protein synthesis efficiency by 200 times Japanese scientists develop ...
Researchers from Cologne, Bochum, Padova and Angers have discovered a novel connection between mitochondrial function, protein quality control and cellular health, whose failure could be the leading ...
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